Poison ivy

Description

Poison ivies can grow as small plants, shrubs, or climbing vines.

File:Poison ivy vine.jpg|T. radicans vine with typical reddish "hairs"

File:Toxicodendron radicans, eastern poison ivy vine, Maryland 2025 01.jpg | Large poison ivy vine

File:Toxicodendron radicans 01.jpg|Flower detail, with bee File:Poison ivy-roadside.jpg|Poison ivy on a roadside File:Poison ivy in May beside Appalachian Trail in Rockfish Gap VA area 1.jpg|Leaves may be smooth or notched on the same plant.

Species

Three species of poison ivy are generally recognised; they are sometimes considered subspecies of Toxicodendron radicans:

• Toxicodendron orientale: found in East Asia.
• Toxicodendron radicans: found throughout eastern Canada and the United States, Mexico and Central America, Bermuda and the Bahamas.
• Toxicodendron rydbergii: found throughout Canada and much of the United States except the southeast.

Toxicity

Main article: Urushiol-induced contact dermatitis

Urushiol-induced contact dermatitis is the allergic reaction caused by poison ivy. In extreme cases, a reaction can progress to anaphylaxis. Around 15 to 25 percent of people have no allergic reaction to urushiol, but most people have a greater reaction with repeated or more concentrated exposure.

Over 350,000 people are affected by urushiol annually in the United States.

The oozing fluids released by scratching blisters do not spread the poison. The fluid in the blisters is produced by the body and it is not urushiol itself. The appearance of a spreading rash indicates that some areas received more of the poison and reacted sooner than other areas or that contamination is still occurring from contact with objects to which the original poison was spread.

Those affected can unknowingly spread the urushiol inside the house, on phones, door knobs, couches, counters, desks, and so on, thus in fact repeatedly coming into contact with poison ivy and extending the length of time of the rash. If this happens, the surfaces should be wiped with bleach or a commercial urushiol removal agent. The blisters and oozing result from blood vessels that develop gaps and leak fluid through the skin; if the skin is cooled, the vessels constrict and leak less. If plant material with urushiol is burned and the smoke then inhaled, this rash will appear on the lining of the lungs, causing extreme pain and possibly fatal respiratory difficulty. If poison ivy is eaten, the mucus lining of the mouth and digestive tract can be damaged.

Urushiol oil can remain active for several years, so handling dead leaves or vines can cause a reaction. In addition, oil transferred from the plant to other objects (such as pet fur) can cause the rash if it comes into contact with the skin.

Treatment

Main article: Urushiol-induced contact dermatitis#Treatments

Immediate washing with soap and cold water or rubbing alcohol may help prevent a reaction. During a reaction, calamine lotion or diphenhydramine may help mitigate symptoms. Corticosteroids, either applied to the skin or taken by mouth, may be appropriate in extreme cases. An astringent containing aluminum acetate (such as Burow's solution) may also provide relief and soothe the uncomfortable symptoms of the rash.

Urushiol binds to the skin on contact where it causes severe itching that develops into reddish inflammation or uncoloured bumps, and then blistering. These lesions may be treated with calamine lotion, Burow's solution compresses, dedicated commercial poison ivy itch creams, or baths to relieve discomfort, though recent studies have shown some traditional medicines to be ineffective. Over-the-counter products to ease itching—or simply oatmeal baths and baking soda—are now recommended by dermatologists for the treatment of poison ivy.

A plant-based remedy cited to counter urushiol-induced contact dermatitis is jewelweed, though jewelweed extracts had no positive effect in clinical studies. Others argue that prevention of lesions is easy if one practices effective washing, using plain soap, scrubbing with a washcloth, and rinsing three times within 2–8 hours of exposure.

The pentadecyl catechols of the oleoresin within the sap of poison ivy and related plants causes the allergic reaction; the plants produce a mixture of pentadecylcatechols, which collectively is called urushiol. After injury, the sap leaks to the surface of the plant where the urushiol becomes a blackish lacquer after contact with oxygen.

Prognosis

Typically, the rash from the urushiol oil lasts about five to twelve days, but in extreme cases it can last a month or more. A urushiol rash usually develops within a week of exposure and can last 1–4 weeks, depending on severity and treatment. In rare cases, urushiol reactions may require hospitalization.

References

References

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